Saturday, December 26, 2009

Penis Enlargement and Theory of How and Why it Works!


by: dickfordays


The penis and it's attachments are made up of several different types of tissue: ligaments, tunica, skin, some muscle, etc. Each type of tissue has a certain amount of short term elasticity. In other words, when I pull, each tissue has a certain amount of give to it before it reaches is maximum short term elasticity. To illustrate, imagine attaching a rubber band to a small weight. If you set the weight on the ground and lift up on the rubber band, the weight will remain on the ground while the rubber band elongates and stretches, until eventually the rubber band reaches is maximum short term elasticity and the weight is finally lifted off the ground (this is assuming the maximum short term elasticity strength is great enough to lift the weight).

A similar situation seems likely to occur when we stretch. Given the various types of tissues that make up the penis and it's attachments, and their individual maximum short term elasticities, an order of stretch must take place. For example, if you tie together several different rubber bands of variable thickness and strength and tie one end to a wall while pulling the other, the most elastic ones are the ones that will stretch and elongate first. The thickest ones may not even stretch at all. In reality, the amount of stretch that each will undergo will be directly related to it's elasticity. In order for the strongest/least elastic band to stretch at all, the tension on the smaller bands will have to be great enough to elongate the stronger bands.

When doing this, the more elastic bands stretch to the point where they become maxed out and rigid before they allow the stretch to be transferred to the stronger bands. In the penis, I assume that the maximum short term elasticity (MSTE) of each component is strong enough to reach it's limit and transfer to the next strongest component (otherwise you would tear something). Also, some components never reach their MSTE because other components come in to play and prevent it long before they could. Skin, for example, is probably capable of stretching a lot more than it gets the opportunity to when stretching because you are stopped by the other tissues. These components, therefore, don't come into play.

All that said, how does this relate to PE? When I have a really good stretching session, I feel the stretch in my ligs for the first few minutes, and then I feel the tension being transferred to my PC area, behind my balls. It seems like the ligs reach their MSTE and become rigid, then the pull transfers to my PC area. I know I have been really maxing out my stretches because my BPFSL and even BPEL have been increasing over the last couple weeks. I theorize that the more you make your ligs (or other components) reach their MSTE and become rigid, the quicker your gains will be. When I feel the stretching burn transfer to my PC area, it seems like an indicator that I have pushed my ligs to their MSTE. At this point in my routine, the burn continues to stay in my PC area regardless of the angle I stretch, even when stretching BTC.

BIB touches on this with his philosophy of hanging. He says that when you hang, the weaker/tighter tissues take the stress first, until they are overcome and break, and then the pull is transferred to the next weakest/tightest tissue, and so on. That's why you may have to periodically reduce the weight you use, because the new limiting tissues cannot handle as much weight at the tissues that were previously the limiting factor (correct me if I'm wrong on this, BIB).

I want to add something to this, in case this applies to anyone here. Regarding short, intense stretching. This makes things more complicated, but I'm afraid it applies.

All materials have properties of viscoelasticity. The viscoelasticity of a material is determined by looking at it's viscous (fluid) properties versus it's elastic properties. VE determines how an elastic material behaves under strain. When a material is stretched and held in a stretched position (under strain) usually one of two things happens: relaxation- the material slackens further making the strain less (like in muscles), or creep- the strain on the material increases (like in connective tissues such as ligaments and tendons). For example, silly putty is considered highly viscoelastic. If you pull it slowly, it will stretch extremely long before breaking (a very fluid property), but if you pull it very quickly, it will break in half almost immediately (a very solid property) because it reaches is elastic limit so quickly when the strain is exerted in such a short period of time. If the rate of stress is much slower, the silly putty stretches out much longer before reaching it's elastic limit (a very elastic property). Therefore, the rate at which the stress is applied will greatly affect the elastic response of a tissue with a high VE.

Ligaments, muscles, tendons, etc. are all also very viscoelastic. This is why short, intense strain to the body results in muscle pulls and ligament/tendon ruptures. The tissues reach their elastic limit when the stress is applied quickly and respond by tearing or rupturing if the force is great enough. However, if you apply the same stress over a longer period of time, such as when stretching your hamstrings, the tissue elongates and stretches. This is one reason why you should never stretch (any kind of stretch) in a quick, jerky manner, but instead ease into a stretch. This brings me to my other point that applies to PE stetching: the Spinal Stretch Reflex.

The Spinal Stretch Reflex can be seen when the doc taps below your knee and your leg jerks. The reason this happens is as follow: All over and within the tissues of the body are proprioceptors. Proprioceptors communicate with the nervous system and act as message relays. They "tell" your body where it is in space and how it is oriented. If you close your eyes and raise your arm up, your body "knows" where your arm is located because of proprioceptors. They also initiate involuntary reflexive actions, such as when your leg jerks. When the doc taps below your knee, he is tapping your patellar tendon that attaches below the knee, and above the knee into your quadriceps. That short tap on the tendon pulls on your quads with a relatively great amount of force, but the force is only applied for a fraction of a second, and is therefore harmless. However, your proprioceptors have already sent the message of the force to your nervous system which has initiated the SSR as a defense mechanism, and the quad contracts to counteract the pull. The SSR is so quick that it does not have time to register the fact that the force is only going to be applied for a fraction of a second, so your quad involuntarily responds.

The SSR is initiated when other tissues of the body are stressed as well. If you apply a short, intense stretch to the penis, the SSR will kick in and the tissues will "pull back" and you will not achieve any stretch. In fact, if you pull hard enough, a tear is possible because of the viscoelastic properties of the tissue. The same amount of force may be harmless if you gradually work up to it and allow the body over-ride the SSR. Any force, no matter how small, initiates the SSR to some degree when initially applied. The reason the tissue lengthens and loosens is because the SSR is over-ridden when the force is not "percieved" as dangerous, by incrementally working up to greater intensities.


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